ارزش تشخیصی ام ار ای در تشخیص افتراقی توده های کبد سیروتیک
ارزش تشخیصی اضافه ام ار ای با پروتکل SWI در تشخیص افتراقی توده های کبد سیروتیک: LIRADS III,LIRADSIV, V
Cirrhosis is a late stage of scarring (fibrosis) of the liver resulting from chronic hepatic inflammation caused by many forms of liver diseases and conditions, such as hepatitis and chronic alcohol abuse, and is characterized by the normal hepatic architecture being replaced with a mixture of parenchymal nodules and fibrosis. The magnetic resonance imaging (MRI) findings of cirrhosis reproduce those histological changes and include altered hepatic morphology, fibrosis, and cirrhotic nodules. The spectrum of cirrhotic nodules includes regenerative nodules, low-grade dysplastic nodules, high-grade dysplastic nodules, and neoplastic nodules.
The Liver Imaging Reporting and Data System (LI-RADS), supported by the American College of Radiology (ACR), provides standardization for hepatocellular carcinoma (HCC) imaging in the contexts of screening and surveillance, diagnosis, and treatment response assessment. LI-RADS was developed by a multinational consortium of radiologists and other specialists with expertise in liver cancer imaging, and it was integrated into the most recent HCC clinical practice guidance by the American Association for the Study of Liver Diseases (AASLD). Cirrhosis is a late stage of scarring (fibrosis) of the liver resulting from chronic hepatic inflammation caused by many forms of liver diseases and conditions, such as hepatitis and chronic alcohol abuse, and is characterized by the normal hepatic architecture being replaced with a mixture of parenchymal nodules and fibrosis. The magnetic resonance imaging (MRI) findings of cirrhosis reproduce those histological changes and include altered hepatic morphology, fibrosis, and cirrhotic nodules. The spectrum of cirrhotic nodules includes regenerative nodules, low-grade dysplastic nodules, high-grade dysplastic nodules, and neoplastic nodules. The development of HCC in a cirrhotic liver is described either as de novo hepatocarcinogenesis or as a multistep progression, from low-grade dysplastic nodule to high-grade dysplastic nodule, then to high-grade dysplastic nodule with microscopic foci of HCC, then to small HCC, and finally to invasive carcin
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